Utilize este identificador para referenciar este registo: http://hdl.handle.net/10451/21065
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degois.publication.firstPage48831por
degois.publication.lastPage48838por
degois.publication.titleJOURNAL OF BIOLOGICAL CHEMISTRYpor
dc.contributor.authorSola, S
dc.contributor.authorMa, XM
dc.contributor.authorCastro, RE
dc.contributor.authorKren, BT
dc.contributor.authorSteer, CJ
dc.contributor.authorRodrigues, CMP
dc.date.accessioned2015-12-30T10:17:26Z-
dc.date.available2015-12-30T10:17:26Z-
dc.date.issued2003
dc.identifier.citationJOURNAL OF BIOLOGICAL CHEMISTRY. - Vol. 278, n. 49 (DEC 5 2003), p. 48831-48838
dc.identifier.issn0021-9258
dc.identifier.urihttp://hdl.handle.net/10451/21065-
dc.description.abstractTransforming growth factor beta1 (TGF-beta1)-induced hepatocyte apoptosis is associated with activation of E2F transcription factors and p53 stabilization through Mdm-2, thus potentially modulating a number of target genes. In previous studies, we have sh
dc.formatapplication/pdf
dc.language.isoeng
dc.publisherAMER SOC BIOCHEMISTRY MOLECULAR BIOLOGY INC
dc.rightsrestrictedAccess
dc.subjectBiochemistry & Molecular Biology
dc.titleUrsodeoxycholic acid modulates E2F-1 and p53 expression through a caspase-independent mechanism in transforming growth factor beta 1-induced apoptosis of rat hepatocytes
dc.typearticle
degois.publication.volumeVol. 278por
dc.identifier.doihttp://dx.doi.org/10.1074/jbc.M300468200
Aparece nas colecções:FF - Produção Científica 2000-2009

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